Comprehensive specialist neurology care for migraine — from the occasional severe attack through to chronic daily migraine. Dr Ron Granot, FRACP, Bondi Junction.
Book a ConsultationMigraine is a neurological disorder, not just a severe headache. It affects approximately 12% of adults — more than diabetes and asthma combined — and is one of the leading causes of disability worldwide, particularly in women aged 25–55. A typical attack lasts 4–72 hours and brings moderate to severe head pain, often one-sided and throbbing, with nausea, vomiting and sensitivity to light and sound. Many patients also experience visual aura, vertigo, brain fog, fatigue and food cravings as part of the attack.
The biology of migraine is now well understood as a brain-based disorder of sensory processing — not muscle tension, sinus pressure or "just stress". It involves cycles of cortical hyperexcitability, trigeminovascular activation, and release of pain-signalling molecules including CGRP. Understanding this matters because it shapes treatment: today we have migraine-specific acute medications (triptans, gepants) and preventatives (Botox, CGRP monoclonal antibodies) that target the actual disorder.
A migraine attack often unfolds in distinct phases. Not every patient experiences every phase, and the timing varies — but recognising the pattern helps with both diagnosis and treatment timing.
Hours to days before head pain. Yawning, mood changes, food cravings, neck stiffness, fatigue, irritability. Often unrecognised until pattern is noticed.
Visual disturbance (zigzag lines, blind spots), sensory changes (tingling), speech disturbance, or weakness. Typically 5–60 minutes before head pain.
4–72 hours of moderate–severe pain, often unilateral and throbbing. Worse with movement. Nausea, photophobia, phonophobia.
Hours to a day or two after. "Migraine hangover" — tired, washed out, mild residual ache, cognitive slowing.
Several distinct migraine variants exist. Recognising them is important because some have specific treatment implications, and some can mimic other neurological problems (or be mimicked by them).
The most common form. Recurrent attacks of moderate–severe headache, often with nausea and light/sound sensitivity, but no preceding aura.
Up to a third of migraine sufferers experience aura — usually visual (zigzag lines, fortification spectra, scotomata). Some have sensory or speech aura. Aura raises specific considerations including stroke risk and contraceptive choice.
≥15 headache days per month, with ≥8 being migraine, for ≥3 months. Specific treatment pathway including PBS-listed Botox and CGRP medications.
Recurrent vertigo or imbalance with migraine features — sometimes without head pain. Commonly misdiagnosed as inner ear disease. See MAV / vestibular migraine.
Rare migraine variant with motor weakness as part of the aura. Can mimic stroke. Familial or sporadic forms. Specific medications are avoided.
Attacks tightly linked to the menstrual cycle, often around the perimenstrual window. Often more severe and longer-lasting. Specific treatment strategies including short-cycle prevention can help.
Several common headache types share features with migraine but call for different treatment. This table gives a quick framework — but the diagnosis is clinical and individual.
| Type | Typical features | Treatment direction |
|---|---|---|
| Migraine | 4–72 h, often one-sided, throbbing, nausea, photophobia, phonophobia, worsens with movement | Acute triptans/NSAIDs/gepants + preventative as needed |
| Tension-type | Bilateral, pressing/tightening, mild–moderate, no nausea, not worsened by routine activity | Simple analgesia, stress management, sometimes amitriptyline |
| Cluster | Severe one-sided around the eye, autonomic symptoms (red eye, tearing, nasal congestion), 15 min–3 h, often nocturnal, cyclical "cluster periods" | Oxygen, subcutaneous sumatriptan; verapamil prevention — see cluster headache |
| Trigeminal neuralgia | Brief (seconds), severe lancinating face pain, triggered by touch/chewing/cold air | Carbamazepine, oxcarbazepine; neurosurgical referral if refractory — see trigeminal neuralgia |
| Medication overuse | Chronic daily headache developing in someone who frequently uses acute medications (≥10 days/month of triptans/codeine; ≥15 days/month simple analgesia) | Structured withdrawal + bridging — see medication overuse headache |
| Cervicogenic | Headache referred from neck structures, often unilateral, made worse by neck movement, restricted neck motion | Physiotherapy, occipital nerve blocks, sometimes facet joint injections |
Most headaches are primary disorders (migraine, tension, cluster). But a small proportion are secondary — caused by another medical problem. The following features should prompt urgent assessment, often in an emergency department rather than an outpatient clinic.
Effective migraine care combines several layers — acute treatment for individual attacks, preventative strategies to reduce frequency, trigger and lifestyle management, and a sustained commitment to finding what works for the individual patient. There is no single "best" treatment, and patience matters.
Book a consultation with Dr Ron Granot — FRACP consultant neurologist, trained at Prince of Wales, Bondi Junction.
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